“Critical contribution of Na+-Ca2+ exchanger to the Ca2+-mediated vasodilation activated in endothelial cells of resistance arteries”. The FASEB Journal. (2018)
“Release of gliotransmitters through astroglial connexin 43 hemichannels is necessary for fear memory consolidation in the basolateral amygdala”. The FASEB Journal. (2012).
“Transient receptor potential melastatin 4 inhibition prevents l i p o p o l y s a c c h a r i d e – i n d u c e d endothelial cell death”.
Cardiovascular Research. (2011)
“Non-Cytotoxic Copper Overload Boosts Mitochondrial Energy Metabolism to Modulate Cell Proliferation and Differentiation in the Human Erythroleukemic Cell Line K562”.
“Excess Iodide Induces an Acute Inhibition of the Sodium/Iodide Symporter in Thyroid Male Rat Cells by Increasing Reactive Oxygen Species”.
“Ezh1 and Ezh2 differentially regulate PSD-95 gene transcription in developing hippocampal neurons”.
Molecular and Cellular Neuroscience. (2013).
“Increases in reactive oxygen species enhance vascular endothelial cell migration through a mechanism dependent on the transient receptor potential melastatin 4 ion channel”.
Microvascular research. (2015).
“Endothelial fibrosis induced by suppressed STAT3 expression mediated by signaling involving the TGF-β1/ALK5/Smad pathway”.
“Lipopolysaccharide induces a fibrotic-like phenotype in endothelial cells”.
Journal of Cellular and Molecular Medicine. (2013).
“Fibrotic response induced by Angiotensin-ii requires NAD(P)H oxidase-induced reactive oxygen species (ROS) in skeletal muscle cells”.
Biochemical and Biophysical Research Communications.(2011).
“The paramagnetic and luminescent [Re6Se8I6]3- cluster. Its potential use as an antitumoral and biomarker agent”.
New Journal of Chemistry. (2012).
“Increased expression of the Transient Receptor Potential Melastatin 7 channel is critically involved in l i p o p o l y s a c c h a r i d e – i n d u c e d ROS-mediated neuronal death”.
Antioxidant & Redox Signalling. 2011.
“Lipopolysaccharide signaling in the carotid chemoreceptor pathway of rats with sepsis syndrome.
Respiratory Physiology and Neurobiology”. (2011).
“Collaboratory for Structural Nanobiology”.
Biophysical Journal. (2009)
“Copper deficiency-induced anemia is caused by a mitochondrial metabolic reprograming in erythropoietic cells.
If you want to know
more about us
We are continuously developing very advanced Ai Solutions to improve patients experiences in healthcare to…. get better